1. Andersson GBJ: The epidemiology of spinal disor- ders, in Frymoyer JW (ed): The Adult Spine: Prin- ciples and Practice, ed 2. New York, NY, Raven Press, 1997, pp 93-141.
  2. Hensinger RN: Spondylolysis and spondylolisthe- sis in children. Instr Course Lect 1983;32:132-151.
  3. Boden SD, Davis DO, Dina TS, et al: Abnormal

    magnetic-resonance scans of the lumbar spine in asymptomatic subjects: A prospective investigation. J Bone Joint Surg Am 1990;72:403-408.

 4. van Tulder MW, Goossens M, Waddell G, et al: Chapter 12: Conservative treatment of chronic
low back pain, in Nachemson AL, Jonsson E (eds): Neck and Back Pain: The Scientific Evidence of Causes, Diagnosis, and Treatment. Philadelphia, PA, Lip- pincott, Williams & Wilkins, 2000, pp 271-304.

5. Weber H: Lumbar disc herniation: A controlled, prospective study with ten years of observation. Spine 1983;8:131-140.

 

Ankylosing spondylitis  An inflammatory disorder that affects the low back and pelvis and produces stiffness and pain

Anulus fibrosus The outer ring of fibrous material surrounding the nucleus pulposus of the intervertebral disks

Cauda equina The terminal nerve roots of the spinal cord located within the vertebral canal; so named because they resemble the tail of a horse

Neurogenic claudication Leg pain that occurs as a result of compression of nerves within the spinal canal; often associated with spinal stenosis

Nucleus pulposus The central core of gelatinous material within intervertebral disks

Osteophytes Overgrowth of bone, common in osteoarthritis and spinal stenosis

Saddle anesthesia Decreased sensation around the perineum

Spinal stenosis Narrowing of the canal housing the spinal cord; commonly caused by encroachment of bone

Spondylolysis A defect or fracture of the pars interarticularis, the portion of bone located between the superior and inferior articular processes of the vertebrae

Spondylolisthesis Anterior displacement of a vertebral body relative to the adjacent vertebral body below

Vascular claudication Leg pain that is caused by ischemia secondary to vascular disease; the pain uniformly occurs with activity and is relieved with rest

 

References

1. Lee DM, Weinblatt ME: Rheumatoid arthritis. Lancet 2001;358:903-911.

  1. Aho K, Koskenvuo M, Tuominen J, et al: Occur- rence of rheumatoid arthritis in a nationwide series of twins. J Rheumatol 1986;13:899-902.
  2. Silman AJ, MacGregor AJ, Thomson W, et al: Twin concordance rates for rheumatoid arthritis: results from a nationwide study. Br J Rheumatol 1993;32: 903-907.
  3. Jenkins RN, Nikaein A, Zimmermann A, et al: T cell receptor V beta gene bias in rheumatoid arthritis. J Clin Invest 1993;92:2688-2701.
  4. Uematsu Y, Wege H, Straus A, et al: The T-cell- receptor repertoire in the synovial fluid of a patient with rheumatoid arthritis is polyclonal. Proc Natl Acad Sci USA 1991;88:8534-8538.
  5. Wagner UG, Koetz K, Weyand CM, et al: Perturba- tion of the T cell repertoire in rheumatoid arthritis. Proc Natl Acad Sci USA 1998;95:14447-14452.
  6. Shiozawa S, Shiozawa K, Fujita T: Morphologic observations in the early phase of the cartilage- pannus junction: Light and electron microscopic studies of active cellular pannus. Arthritis Rheum 1983;26:472-478.
  7. McCachren SS, Haynes BF, Niedel JE: Localization of collagenase mRNA in rheumatoid arthritis synovium by in situ hybridization histochemistry.J Clin Immunol 1990;10:19-27.

    9. Gravallese EM, Darling JM, Ladd AL, et al: In situhybridization studies of stromelysin and collage- nase messenger RNA expression in rheumatoid synovium. Arthritis Rheum 1991;34:1076-1084.

    10. Martel-Pelletier J, McCollum R, Fujimoto N, Obata K, Cloutier JM, Pelletier JP: Excess of metallopro- teases over tissue inhibitor of metalloprotease may contribute to cartilage degradation in osteoarthri- tis and rheumatoid arthritis. Lab Invest 1994;70:807- 815.

    11. Bland JH: Rheumatoid arthritis of the cervical spine. J Rheumatol 1974;1:319-342.

    12. Kauppi M, Sakaguchi M, Konttinen YT, et al: Pathogenetic mechanism and prevalence of the stable atlantoaxial subluxation in rheumatoid arthritis. J Rheumatol 1996;23:831-834.

    13. McCarty DJ: Gout without hyperuricemia. JAMA 1994;271:302-303.

    14. Lawry GV II, Fan PT, Bluestone R: Polyarticular versus monoarticular gout: A prospective, com- parative analysis of clinical features. Medicine (Baltimore) 1988;67:335-343.

    15. Emmerson BT: The management of gout. N Engl J Med 1996;334:445-451.

Gout – An inflammatory arthritis associated with deposition of urate crystals in the joint

Hyperuricemia – Elevated serum levels of urate

Juvenile rheumatoid arthritis – A chronic inflammatory disease in children that is characterized by pain, swelling, and tenderness in one or more joints and may result in impaired growth and development

Rheumatoid arthritis – A chronic inflammatory disease that is probably triggered by an antigen-mediated inflammatory reaction against the synovium in the joint

Tophi – Chalky deposits of urate surrounded by inflammatory cells

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